Revolutionary Alzheimer’s disease study points to vitamin C as a universal nutrient for prevention
12/19/2017 // Tracey Watson // Views

Alzheimer’s disease is the scourge of the elderly. Characterized by mental decline, difficulty understanding and thinking, confusion, forgetfulness, disorientation, loss of memories and an inability to create new ones, and the inability to recognize common things, among other devastating symptoms, Alzheimer’s has no known cure. This is one of the most devastating illnesses for families to cope with, as they literally watch their loved ones disappear slowly before their eyes.

There is no known cure for Alzheimer’s and the causes of the condition are poorly understood.

Now, an exciting new study by researchers from the Vanderbilt University School of Medicine offers real hope for the prevention of Alzheimer’s. The study was published in the journal Free Radical Biology and Medicine.

The study abstract explains that Alzheimer’s is associated with alterations in mitochondrial function which appear even before the earliest symptoms become noticeable. This dysfunction increases production of reactive oxygen species (ROS), which in turn cause the increased production of Amyloid beta. This pattern accelerates the development of Alzheimer’s disease.

The authors note that vitamin C is a potent antioxidant, and their study set out to determine if it could protect the brain against this damaging pattern. (Related: Discover the amazing healing potential of nature’s medicine cabinet at Nutrients.news.)

The researchers used mouse models to determine the effects of vitamin C deficiency and Alzheimer’s-related mutations on the functioning of the mitochondria.

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Vanderbilt reported on the results of the study:

Vitamin C deficiency led to diminished mitochondrial respiration and increased ROS, while mitochondria from the mouse model of Alzheimer’s displayed increased respiration compared to wild type controls. The results suggested that both vitamin C deficiency and the presence of amyloid contribute to mitochondrial dysfunction but via differing pathways.

These findings … suggest that vitamin C deficiency could contribute to the pathogenesis of Alzheimer’s through altered mitochondrial function and that avoiding deficiency through diet and supplementation could protect against disease onset.

While this study offers hope for the prevention and treatment of Alzheimer’s disease, there are many other reasons to supplement your diet with a source of good quality vitamin C.

Vitamin C has been found to reduce the risk of developing heart disease, which is the world’s number one killer. It has also been shown to annihilate cancer. And it outperforms vaccines when it comes to boosting immunity and preventing disease – without the deadly side effects associated with those toxic jabs.

What many of us are unaware of, however, is that vitamin C’s potent antioxidant power prevents oxidative stress to cells throughout the body, literally healing the body from the inside out and promoting a longer, healthier life.

A study published in the American Journal of Clinical Nutrition, based on data collected from over 10,000 people living in Denmark, found that those with the highest levels of vitamin C in their bloodstreams were the least likely to develop heart disease or die prematurely.

Vitamin C also helps the body produce collagen, which regenerates and protects muscles, tendons, ligaments and the connective tissues in the body which maintain the health of important organs like the heart, liver and pancreas.

Natural News previously reported:

Other important functions of vitamin C in the body include the promotion of wound healing, the production of the blood vessels which carry oxygen and nutrients all over the body and even with the regulation of moods and emotions. Taken all and all, then, it is no surprise that high vitamin C levels can lead to a longer life.

From Alzheimer’s prevention to living a longer, healthier life, there are certainly sound reasons for adding more vitamin C to your daily diet.

Sources for this article include:

News.Vanderbilt.edu

NCBI.NLM.NIH.gov

NaturalNews.com



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