Originally published September 7 2005
Cell mitochondria defect and diabetes linked, study shows
by Mike Adams, the Health Ranger, NaturalNews Editor
A study by the Yale School of Medicine shows difficulty burning fats and sugars may be a major factor in the development of diabetes and suggests the problem lies in cell mitochondria, the cell's energy sources.
Senior author Dr. Gerald Shulman, of the Yale School of Medicine, said in a new study a decreased ability to burn sugars and fats efficiently is an early and central part of the diabetes problem.
The new data suggest the basic defect lies within the mitochondria, which are the energy factories inside cells that produce most of the chemical power needed to sustain life.
The researchers observed the mitochondria in the subjects' muscle cells responded poorly to insulin stimulation, but normal mitochondria react to insulin by boosting production of an energy-carrying molecule, ATP, by 90 percent.
The mitochondria from the insulin-resistant people only boosted ATP production by five percent.
The findings are published in PLoS Medicine 2.
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