Robert Anton Wilson
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 And we can never have pure neurology but only neuro-neurology ?neurology as known through the human nervous system...
But at this point we have already entered the arena of Strange Loops, as some readers have guessed, for neuro-neurology can only be known by the nervous system and thus can only be known by a meta-science of neuro-neuro-neurology...which can only be known through neuro-neuro-neuro-neurology...and so on, ad infinitum. Do you detect Lord Russell's two-head argument looming on the horizon at this point? Or even T. W. Dunne's infinite regress of consciousnesses in time? |
Peter J. Whitehouse and Daniel George
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 As the historian Jesse Ballenger has pointed out, from
91
1926 to 1935 the American Journal of Psychiatry and the Archives of neurology and Psychiatry, the two leading professional journals of American psychiatry and neurology, ran only nine articles concerning Alzheimer's disease.1 In the middle of the twentieth century, little mention was made of AD, and only a small number of clinicians used the label, reserving it only for patients in their fourth, fifth, or sixth decade who showed the clinical and neuropathological signs of senile dementia. |
Dr. Arthur Janov
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 Until now, there has been no theoretical web that encompasses both psychology and neurology, although there have been attempts to join psychoanalysis with neurology. It is, by and large, a shotgun wedding. It is the same as plastering an old, outdated notion onto new science and hoping it will stick. If psychoanalysis ignores key internal realities, it doesn't matter that we adhere certain neurologic facts to it. It cannot work. Why would we take a theory that is 100 years old and join with it research that may be six months old? |
Steven V. Joyal
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 These data can be contrasted with recently published data in the scientific journals Archives of neurology and Experimental neurology, which showed that much higher doses of ubiquinone were required to obtain comparable blood CoQIO levels. Specifically, the studies with ubiquinone used 1,200 mg per day to achieve blood concentrations of 3.96 mcg/mL and 2,400 mg per day to reach blood levels of 7.25 mcg/mL.
Scientists have long known that CoQl 0 levels decrease with age and that there is a clear association between that decline and age-related problems related to energy production in cells. |
Dr. Abram Hoffer, MD, FRCP (C) and Dr. Harold D. Foster, PhD
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 Annals of neurology,i989;25(4):3i7-24.
9 Hoffer A, Walker M. Smart Nutrients: Prevent and Treat Alzheimer's and Senility, Enhance Brain Function and Longevity. Ridgefield, CT: Vital Health Publishing, 2002.
10 Hoffer A. Niacin Therapy in Psychiatry. Springfield, IL: CC Thomas, 1962.
11 Morris MC, Evans DA, Bienias PA, Scherr A, Tangney CC, Hebert LE, Bennett DA, Wilson RS, Aggarwal N. Dietary niacin and the risk of incident Alzheimer's disease and of cognitive decline. J neurology, Psychiatry 2004;75:1093-99.
12 Foster HD. What Really Causes Alzheimer's Disease. |
Mark Sircus
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 Statins and risk of polyneuropathy
D Gaist, MD PhD, U Jeppesen, M Andersen, LAG neurology 2002;58:1333-1337 ?2002 American Academy of neurology Statins and risk of polyneuropathy.
65 Participants receive injections of the toxin in six places in the calf muscle and then the leg is put into a cast. The idea is that this will help prevent pressure on the ball of the foot during walking. The ball if the foot is the area most affected by foot ulcers and allowing an ulcer to heal completely helps prevent recurrence, (www.diabetes-and-diabetics.com/about-diabetes/diabetic-complications-02. |
Steven V. Joyal
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| These data can be contrasted with recently published data in the scientific journals Archives of neurology and Experimental neurology, which showed that much higher doses of ubiquinone were required to obtain comparable blood CoQIO levels. Specifically, the studies with ubiquinone used 1,200 mg per day to achieve blood concentrations of 3.96 mcg/mL and 2,400 mg per day to reach blood levels of 7.25 mcg/mL.
Scientists have long known that CoQl 0 levels decrease with age and that there is a clear association between that decline and age-related problems related to energy production in cells. |
Dr. Abram Hoffer, MD, FRCP (C) and Dr. Harold D. Foster, PhD
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| Dietary niacin and the risk of incident Alzheimer's disease and of cognitive decline. J neurology, Psychiatry 2004;75:1093-99.
12 Foster HD. What Really Causes Alzheimer's Disease. Victoria, BC: Trafford Publishing, 2004.
J3 Hoffer A. Niacin Therapy in Psychiatry. Springfield, IL: CC Thomas, 1962.
14 Hoffer A. Senility is a form of chronic malnutrition. Report of a National Conference on The Crisis in Health Care for the Aging, sponsored by the Huxley Institute of Biosocial Research, New York, March 6,1972.
15 Wysong, P. High HDL cholesterol may protect against dementia. |
Shannon Brownlee
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 The lightbulb moment for me was when I got the neurology intern to explain to me why Mr. S. was in four-point restraints," she says. "The intern looked at me with enormous distress in his eyes and he said, 'He'll pull out the nasogastric tube. If I don't keep him in restraints, he'll die.' "
It might seem like a giant leap to go from the suicide of a young college student to the death of an elderly man riddled with cancer, yet Justin Cheslek and Mr. S. are linked to each other by the American belief that more is better when it comes to medicine. |
| In 200 c, the Journal of the American Medical Association published a survey of 824 Pennsylvania doctors in high-risk specialties, including obstetrics, neurology, and emergency medicine; 59 percent of those surveyed said they routinely ordered unnecessary tests, including imaging tests. Doctors know perfectly well they are ordering useless imaging tests, but when you ask them why they do it, they offer conflicting reasons. |
Donna Jackson Nakazawa
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 One 2006 study reported in the Archives of neurology, published by the American Medical Association, shows that young adults who have high levels of antibodies against the Epstein-Barr virus are significantly more likely to develop multiple sclerosis fifteen to twenty years down the road. Harvard School of Public Health and Kaiser Permanente researchers were able to determine this correlation by examining records of more than 100,000 patients who joined health plans between 1965 and 1974, when they were in their early thirties. |
Thomson Healthcare, Inc.
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 New onset migraine associated with use of Soy isoflavone supplements. neurology 59(8):1289-1290. 2002.
Erdman JW. Control of serum lipids with Soy protein. N Engl J Afe?333(5):313-315. 1995.
Erdman JW. Soy protein and cardiovascular disease, a statement for healthcare professionals from the nutrition committee of the AHA. Circulation 102!(20):2555-2559. 2000.
Fair WR, Fleshner NE, Heston W. Cancer of the prostate: a nutritional disease? Urology 50(6):840-848. 1997.
Faure ED, Chantre P, Mares P. |
Shannon Brownlee
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| The hospital admitted him to the neurology department, and the doctors stabilized his condition with drugs that reduced the swelling of his brain, but it was only a matter of days before the cancer killed him. Mr. S. went in and out of awareness, but his son, who was serving as his health care proxy, knew he did not want to be treated for his cancer.
When Meier entered the room, she found a frail, emaciated man who was unable to speak intelligibly but was nonetheless clearly agitated, thrashing around in his bed despite being pinned by his wrists and ankles in four-point restraints. |
Donna Jackson Nakazawa
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| Douglas Kerr, associate professor of neurology at the Johns Hopkins University School of Medicine, and principal investigator on this groundbreaking stem-cell study that recently rocked the scientific world. Whether by disposition or by training, Kerr is reluctant to accept the wave of admiration coming his way. He lowers his hands to stem the tide of applause, eager to explain to the crowd how, utilizing embryonic mouse stem cells in a novel set of strategic scientific steps, he has been able to regenerate the damaged axonal nerves and myelin sheaths in paralyzed rats. |
| You might remember that this is the same inflammatory cytokine that Douglas Kerr, associate professor of neurology at Johns Hopkins School of Medicine, found in elevated levels in the spinal fluid of MS patients. Indeed, Kerr found that in patients with MS and transverse myelitis, the level of IL-6 proteins in their blood closely correlated with the severity of their paralysis. The same cytokine activity now being measured to help diagnose MS is also present in higher levels in those going through chronic stress.
Of course, one might ask which came first, the chicken or the egg? |
Shannon Brownlee
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| Even so, head CTs transformed the practice of emergency medicine and neurology. Before
CT, patients who came into the emergency department with trauma to the head were X-rayed, which could show skull fractures but not bleeding inside the skull. By the time doctors realized the patient was bleeding and sent her in for surgery, it was often too late to prevent devastating brain damage, or even death. |
Donna Jackson Nakazawa
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| Ahmet Hoke, associate professor and director of the Division of Neuromuscular Diseases of the Department of neurology and Neuroscience. In his lab on the fifth floor of the pathology building of Johns Hopkins Hospital, Hoke—who is also my own neurologist—recently discovered that a unique growth factor produced naturally by our nerve cells has a surprising ability to regenerate nerves in the body after they've been damaged by diseases such as multiple sclerosis, transverse myelitis, Guillain-Barre syndrome, and chronic inflammatory demyelinating polyneuropathy. |
Thomson Healthcare, Inc.
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| Acute neurologic events associated with ingestion of Ma Huang (Ephdera). Neurology; 46:A282-A283. 1996.
Gazaliev AM, Fazilov SD, Zhurinov MZ, Khim Prorod Soed 23:862-864. 1987.
Goldberg LI. Monoamine oxidase inhibitors: adverse reactions and possible mechanisms. JAMA; 190:456-462. 1964.
Greenway FL, De Jonge L, Blanchard D, et al. Effect of a dietary herbal supplement containing caffeine and ephedra on weight, metabolic rate, and body compostion. Obes Res; 12(7): 1152-1157. 2004.
Gulati OD, Dave BT, Gokhale SD et al. Antagonism of adrenergic neuron blockade in hypertensive subjects. |
Anne Harrington
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 In the German-speaking countries (Germany, Austria, Switzerland), psychosomatic medicine institutionalized itself in the 1920s in ways that kept it closely allied with the professions of nerve doctoring and neurology (one of the key vehicles for the new ideas was the journal Der Nerven-arzt, founded in 1928). At the same time, the field increasingly identified its cause with larger efforts in German-speaking European medicine to challenge mechanistic, dualistic, and reductionistic thinking and to turn medical practice in general in more holistic and naturopathic directions. |
Fred A. Baughman, Jr., M.D. and Craig Hovey
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 After graduating I went on to specialize in neurology, which I practiced in Grand Rapids, Michigan, then San Diego, California. In addition to a busy private practice of adult and child neurology, I maintained an active presence in neurological research, where I discovered and verified several neurological and genetic diseases.
During the 60s and 70s I became concerned with the increasing numbers of psychiatric and learning disorders children were being diagnosed with. |
Anne Harrington
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| Nevertheless, he also acknowledged equally significant allegiances to Gestalt psychology, clinical neurology, and the existentialist philosophies of both Martin Heidegger and the Jewish theologian Martin Buber (a close friend).36
Weaving all these influences together, Weizsacker's psychosomatic medicine took as its starting point the principle that the patient should be at the center of every clinical encounter, not as an object of medical investigation but as an experiencing subject. |
Jonny Bowden, Ph.D., C.N.S.
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 One study done in 1991 and published in neurology divided 130 Alzheimer's patients into two groups. One was treated with acetyl-L-carnitine while the other received a placebo. In thirteen of the fourteen outcome measures (including long-term verbal memory, selective attention, and logical intelligence), the acetyl-L-carnitine group had better scores. Those patients who had "good treatment compliance"—meaning they actually listened to the doctor and took their supplements on a regular basis—showed even greater benefit. |
| In an article in the prestigious New England Journal of Medicine, researchers from the department of neurology at Boston University School of Medicine found that a high level of homocysteine in the blood "is a strong, independent risk factor for the development of dementia and Alzheimer's disease." Homocysteine levels are easily brought back down by vitamin B12, vitamin B6, and folic acid. It is quite common for the elderly to be deficient in B12. |
Paul D. Blanc, M.D.
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 This was a period when neurology was clearly establishing itself as a distinct discipline; psychiatty, as such, did not exist yet.
Consistent with a general biological model of disease, in which toxin-based examples were considered particularity valuable for their mechanistic insights, poisons to the nervous system (neurotoxins) were especially noteworthy to the clinicians of Delpech's day. This was all the more true in the study of mental derangement, a mechanistically impenetrable group of neurological conditions, ranging from melancholy to madness. |
Andreas Moritz
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 Another study published in the Lancet neurology (May 1, 2007) showed that seniors who regularly take regular aspirin and warfarin to prevent strokes could instead actually be increasing their risk. Especially in healthy older people over 75, aspirin may very well be doing more harm than good. The number of strokes associated with these drugs increased by a factor of seven. The researchers concluded that the increasing use of these drugs means that they may soon overtake high blood pressure as the leading cause of intracerebral hemorrhagic stroke in those over 75. |
| This finding was reported in the Archive Of neurology (Volume 62, page 1539).
More evidence has been uncovered that Alzheimer's disease may actually be a third form of diabetes, according to researchers from Northwestern University (Chicaco). Insulin and insulin receptors in your brain are crucial for learning and memory. For this reason, the brain makes its own insulin. In 2005, it was discovered that both insulin and insulin receptors are lower in people with Alzheimer's disease. |
Bill Sardi
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 Journal neurology Neurosurgery Psychiatry 75: 1636-38, 2004]
¦ Eye doctors have found tuberculosis masquerading as ocular tumors. [Survey Ophthalmology 49: 619-20, 2004]
¦ In Poland, researchers report the case of a 47-year-old man with Hodgkin's lymphoma and enlarged lymph nodes in his neck. After tests were conducted, no viruses could be found. The insertion of a scope into the patient's lungs to obtain a tissue sample resulted in the discovery of tubercular bacteria. |
| Urology International 75:340-4, 2005; J Clinical Oncology 23:8570-9, 2005; neurology 64:26-31,2005]
Dr. Kedar N. Prasad, at the Center for Vitamin and Cancer Research, Department of Radiology, University of Colorado Health Sciences Center, Denver, says most oncologists do not recommend antioxidants to their patients during standard therapy, but some may recommend them at low doses after completion of therapy. However, about 60% of patients take antioxidants during standard therapy without the knowledge of their oncologists. |
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