Mike Adams, the Health Ranger See article keywords and concepts | Aloe vera saves animals and human from severe hemorrhagic shock (blood loss)
Here's a fascinating use of phytonutrients found in aloe vera. I've talked about this in previous articles but never discussed it in detail until now. It turns out that special polymers found in aloe vera gel have the ability to potentiate the oxygen carrying capacity of blood.
What do I mean by that? I mean that aloe vera gel turns sludgy, oxygen-depleted blood into smooth-flowing oxygen-rich blood! It's being used experimentally right now by the U.S. | Michael Friedman, ND See book keywords and concepts | Increases also occur in hypoglycemia and hemorrhagic shock.
The exact connection between schizophrenia and oxidized catecholamine metabolites has not been studied. It is unlikely they are specific to schizophrenia, most likely due to a combination of factors, such as a deficiency of reducing substances or antioxidants, an excess of oxidizing metals or enzymes, and an overload of catecholamines. The increased amount of adrenochrome and like substances cannot be detoxified fast enough, leading to an accumulation of adrenochrome and its metabolites. | Gary Null, Ph.D. See book keywords and concepts | Hemorrhagic Shock
Results of this study found that the administration of superoxide dismutase enhanced the lifespan of rats exposed to hemorrhagic shock.
—L.R. Tan, et al., "Superoxide Dismutase and Allopurinol Improve Survival in an Animal Model of hemorrhagic shock," Am Surg, 59(12), December 2993, p. 797-800.
Ischemic Spinal Cord Injury
Results of this study showed that treatment with superoxide dismutase reduced motor dysfunction and spinal infarcts a week following ischemia in rabbits.
—P. Cuevas, et al. | The Life Extension Editorial Staff See book keywords and concepts | Innovative Drug Strategies Hydergine
Hydergine, an antioxidant medication that helps to protect brain cells, may be beneficial for the treatment of hemorrhagic shock. In Europe, Hydergine is administered on an acute-care basis for the prevention of brain damage following stroke. The recommended dosage of Hydergine in an acute situation is 10 mg administered sublingually and 10 mg given orally. Because the FDA has not approved Hydergine for use in the treatment of stroke, emergency room physicians may not be willing to administer this medication. | | The medications Hydergine and piracetam may be beneficial to patients with hemorrhagic shock. The FDA has not approved Hydergine for the treatment of stroke, but it should be available through the hospital pharmacy, and patients or their surrogates should request its use. Piracetam may be beneficial in preventing permanent neurological damage following stroke. Piracetam is not currently available in the United States.
There is little research on natural supplements for hemorrhagic stroke. CDP-Choline and vitamin C may be of some benefit in facilitating recovery and preventing future strokes. | Gary Null, Ph.D. See book keywords and concepts | This study examined the of endogenous glutathione in gastric mucosal injury associated with hemorrhagic shock and reperfusion. Results showed that the glutathione protected against mucosal ischemia/ reperfusion injury and prevented the ischemia/reperfusion-induced drop in mucosal glutathione.
—H.J. Stein, et al., "Gastric Mcosal Injury Caused by hemorrhagic shock and Reperfusion: Protective Role of the Antioxidant Glutathione," Surgery, 108(2), August 1990, p. 467-473. | Gary Null, Ph.D. See book keywords and concepts | Superoxide dismutase enhanced the life span of rats exposed to hemorrhagic shock. Treatment with SOD reduced motor dysfunction and spinal infarcts a week following ischemia in rabbits. Superoxide dismutase has exhibited significant protective effects against kidney damage induced by ischemia and reperfusion in dogs and rats. Glycosulated SOD derivates, galactosylated and mannosylated, prevented ischemia/reperfusion-induced liver damage in rats. Superoxide dismutase has protected against lung damage in numerous animal trials. |
Textbook of Natural Medicine 2nd Edition Volume 1Michael T. Murray, ND See book keywords and concepts | | Superoxide dismutase polyethylene glycol improves survival in hemorrhagic shock. Am Surg 1991; 57: 747-750
34. Giri SN, Misra HP. Fate of superoxide dismutase in mice following oral route of administration. Med Biol 1984; 62: 285-289
35. Zindenberg-Cherr S, Keen CL, Lonnerdal B, Hurely LS. Dietary superoxide dismutase does not affect tissue levels. Am J Clin Nutr 1983; 37: 5-7
36. Bucci LR, Klenda BA, Stiles JC. Nutritional supplements containing antioxidant enzymes: label claims and potencies. | Sheldon Saul Hendler and David Rorvik See book keywords and concepts | Glutamine dipeptide-supplemented parenteral nutrition reverses gut mucosal structure and interleukin-6 release of rat intestinal mononuclear cells after hemorrhagic shock. Shock. 1998; 10:26-31.
Glutathione
DESCRIPTION
The term glutathione is typically used as a collective term to refer to the tripeptide L-gamma-glutamyl-L-cysteinylglycine in both its reduced and dimeric forms. Monomeric glutathione is also known as reduced glutathione and its dimer is also known as oxidized glutathione, glutathione disulfide and diglutathione. | Alan Keith Tillotson, Ph.D., A.H.G., D.Ay. See book keywords and concepts | A study on rabbits suffering from hemorrhagic shock examined the effects of various combinations of salvia root, tien chi root, and chuan xiong rhizome (Ligusticum wallichii). Blood tests showed that all three herbs were effective for relieving blood pressure and heart rate reduction but that the combination of any two herbs was superior to using a single herb, improving results and lowering the required dosage (Wang etal., 1997).
TULSI
Latin: Ocimum sanctum Sanskrit: Tulsi English: Holy basil
WHAT IT DOES: Tulsi is bitter in taste and cold in action. | Gary Null, Ph.D. See book keywords and concepts | Gastric Mcosal Injury Caused by hemorrhagic shock and Reperfusion: Protective Role of the Antioxidant Glutathione," Surgery, 108(2), August 1990, p. 467-473.
General
Results of this study demonstrated a causal relationship between glutathione transport and protection from oxidative injury in type II cells from neonatal and fetal rats.
—L.A. Brown, et al., "Glutathione Protection in Alveolar Type II Cells from Fetal and Neonatal Rabbits," American Journal of Physiology, 262(3 Pt 1), March 1992, p. L305-12. | | Results of this study showed that melatonin restored the depressed immune function following soft-tissue trauma and hemorrhagic shock in mice.
—M.W. Wichmann, et al., "Melatonin Administration Attenuates Depressed Immune Functions Trauma-Hemorrhage," Journal of Surgical Research, 63(1), June 1996, p. 256-262.
Results of this study showed that the chronic injection of melatonin into either young mice or those suffering from aging-induced immunodepression or immunodepression due to treatment with cyclophosphmide enhanced antibody response to a T-dependent antigen.
—M.C. Caroleo, et al. | | Superoxide Dismutase and Allopurinol Improve Survival in an Animal Model of hemorrhagic shock," Am Surg, 59(12), December 2993, p. 797-800.
Ischemic Spinal Cord Injury
Results of this study showed that treatment with superoxide dismutase reduced motor dysfunction and spinal infarcts a week following ischemia in rabbits.
—P. Cuevas, et al., "Ischemic Reperfusion Injury in Rabbit Spinal Cord: Protective Effect of Superoxide Dismutase on Neurological Recovery and Spinal Infarction," ActaAnat, 137(4), 1990, p. 303-310. | | Results of this study showed that melatonin restored the depressed immune function following soft-tissue trauma and hemorrhagic shock in mice.
—M.W. Wichmann, et al., "Melatonin Administration Attenuates Depressed Immune Functions Trauma-Hemorrhage," Journal of Surgical Research, 63(1), June 1996, p. 256-262.
Results of this study showed that the chronic injection of melatonin into either young mice or those suffering from aging-induced immunodepression or immunodepression due to treatment with cyclophosphmide enhanced antibody response to a T-dependent antigen.
—M.C. Caroleo, et al. |
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